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Nephrology 2

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What is the differential for non-anion gap metabolic acidosis? (DURHAM mnemonic)
Diarrhea, Ureteral diversion, RTA, Hyperalimentation, Acetazolamide, Misc (Ampho B)
What is Winter's formula and when should it NOT be used?
[1.5(HCO3) + 8] +/- 2. Should not be used if primary disorder is not metabolic acidosis.
Why does a metabolic alkalosis ensure from volume contraction?
Chloride is depleted in volume contraction and since the kidenys become sodium avid, HCO3- is reabsorbed with sodium.
What should be the next test to order after a renal panel and ABG in metabolic alkalosis?
Urinary chloride.
What are the causes of saline-responsive metabolic alkalosis?
NG suction, Vomiting, diuretics, post-hypercapnea
What are the causes of saline-RESISTANT metabolic alkalosis with a normal BP?
Hypomagnesemia, severe hypokalemia, Bartter's, NaHCO3 administration, licorice
What are the causes of saline-RESISTANT metabolic alkalosis with a ELEVATED BP?
Conn's, Cushing's, RAS,
How much should the PC02 rise for every 1 increase in HCO3 above 24?
0.5 - 1
For every 10 increase in PCO2 what is expected changes in pH and HCO3? Acutely? Chronically?
ACUTE: pH falls 0.08 and HCO3 increases by 1. CHRONIC: pH falls by 0.03 and HCO3 increases by 3-4.
Which RTA shows an inability to acidify the urine below 5.5 despite an acid (NH4Cl) challenge?
RTA I
What is the most common cause of type I RTA?
congenital autosomal dominant disorder
What genetic disorders are associated with type I RTA?
Ehrlers-Danlos; Marfan's
What autoimmune diseases are classically associated with type I RTA?
SLE, RA, Sjogrens
What medications are associated with a type I RTA?
Amphotericin, lithium, high dose salicylates.
What are the essential defects in RTA types I, II and IV?
Type I: inability to excrete H+ in the distal tubule; Type II: inability to resorb HCO3 in the prox tubule; Type IV hyporeninemic hypoaldosteronism
What hemoglobinopathy and anatomic urinary pathology lead to RTA, type I?
sickle cell anemia and obstructive uropathy.
What disorders of calcium homeostasis occur in RTA, type I?
hypercalciuria and calcium oxalate stones. Hyperparathyroidism and vit D deficiency may develop leading to rickets or osteomalacia.
What confirmatory test is used to diagnose RTA, type I?
Acid infusion (NH4Cl). Fails to acidify urine.
What are causes of RTA, type II?
Fanconi's, carbonic anhydrase inhibitors, outdated tetracycline, lead and mercury toxicity.
What are some other causes of type I RTA?
hepatitis (cirrhosis), nephrocalcinosis, and multiple myeloma
What type of RTA does multiple myeloma cause?
Types I & II
What are some causes of RTA type II?
Fanconi's, amyloidosis, multiple myeloma, acetazolamide
What are some causes of RTA type IV?
Diabetic nephropathy (most common), NSAIDs, ACEIs, ARBs, heparin, TMP-SMX, K+ sparing diuretics, pentamadine, cyclosporine A, tacrolimus, Addisons
Which drugs are associated with RTA?
Type II: outdated tetracycline, acetazolamide; Type I: amphotericin B, lithium, high dose salicylates; Type IV: NSAIDS, heparin, TMP-SMX, pentamadine, cyclosporine A, tacrolimus, ACEI/ARB, K+ sparing diuretics
What is the calculation for serum osmolarity?
[(2xNa)+(BUN/2.8)+(glc/18)+(EtOH/4.6)]
What percentage of weight is TBW and what percentage is typically intracellular vs. extracellar?
TBW is 60% of weight and 2/3 is intracellular
What is the average "insensible" water loss/day?
1L
What are the main branch points in assessing hyponatremia?
1) rule out pseudohyponatremia, 2) measure serum osmolarity to confirm hypotonic hyponatremia, 3) assess volume status, 4) Check urine sodium
How do you calculate free water deficit?
pt wt x 0.4-0.6 x (ptNa/140 -1)

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